World Library  


Add to Book Shelf
Flag as Inappropriate
Email this Book

Plos One : Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in Dy2J Mouse Model of Congenital Muscular Dystrophy, Volume 8

By Gillingwater, Thomas, H.

Click here to view

Book Id: WPLBN0003964149
Format Type: PDF eBook :
File Size:
Reproduction Date: 2015

Title: Plos One : Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in Dy2J Mouse Model of Congenital Muscular Dystrophy, Volume 8  
Author: Gillingwater, Thomas, H.
Volume: Volume 8
Language: English
Subject: Journals, Science, Medical Science
Collections: Periodicals: Journal and Magazine Collection (Contemporary)
Historic
Publication Date:
Publisher: Plos

Citation

APA MLA Chicago

Gillingwater, T. H. (n.d.). Plos One : Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in Dy2J Mouse Model of Congenital Muscular Dystrophy, Volume 8. Retrieved from http://netlibrary.net/


Description
Description : Omigapil therapy (0.1 mg/kg) improved respiratory rate and decreased skeletal and respiratory muscle fibrosis in dy2J mice. These results support a putative role for the use of omigapil in laminin deficient congenital muscular dystrophy patients.

 

Click To View

Additional Books


  • Plos One : Elevated Pretherapy Serum Il1... (by )
  • Plos One : Relationship Between Affectiv... (by )
  • Plos One : a Texture Based Pattern Recog... (by )
  • Plos One : Distinct Features of Auditory... (by )
  • Plos One : Ventral Striatal Activity Cor... (by )
  • Plos One : Larval and Post-larval Stages... (by )
  • Plos One : Effects of Nucleoside Analogu... (by )
  • Plos One : Sumoylation of Gtf2Ird1 Regul... (by )
  • Plos One : Improvement of the Vietnamese... (by )
  • Plos One : Effect of Parenteral Selenium... (by )
  • Plos One : Small Gtpase Rab40C Associate... (by )
  • Plos One : Individual Variation in Level... (by )
Scroll Left
Scroll Right

 



Copyright © World Library Foundation. All rights reserved. eBooks from World Library are sponsored by the World Library Foundation,
a 501c(4) Member's Support Non-Profit Organization, and is NOT affiliated with any governmental agency or department.